Acute Pediatric Neurology by Thomas Sejersen, Ching H. Wang PDF

By Thomas Sejersen, Ching H. Wang

ISBN-10: 0857294903

ISBN-13: 9780857294906

This e-book offers strategies for overview and remedy within the zone of acute pediatric neurology; those are offered didactically with widespread use of illustrations and algorithms. Chapters within the first a part of the publication speak about offering indicators of acute neurological stipulations. the second one a part of the booklet covers significant components of acute pediatric neurology and every of those chapters has 3 key components: description of proposing signs; steered tests; and advised interventions.

 

Acute Pediatric Neurology provides an available, clinically centred consultant to help physicians within the emergency ward or in depth care unit in judgements on prognosis and healing interventions in all significant acute pediatric neurological diseases.

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Additional info for Acute Pediatric Neurology

Sample text

This decreased benzodiazepine response occurs rapidly after the onset of SE in young animals with ages corresponding to a human toddler [15]. Diazepam and the other benzodiazepines enhance the function of a subset of benzodiazepinesensitive GABAA receptors. The surface expression of these receptors declines during SE resulting from activity-dependent trafficking of subunits of GABAA receptors [12]. J. Riviello Jr. C. 3 Generalized seizures Tonic-clonic (any combination) Absence Typical Atypical Absence with special features: Myoclonic Myoclonic atonic Myoclonic tonic Clonic Tonic Atonic Focal seizures Unknown Epileptic spasms Presenting Acute Symptoms The classification of the seizure starts with the seizure semiology, defined as the study of signs and symptom with seizure types divided into either focal or generalized seizures.

Neuroimaging (CT, MRI) findings correlating with poor outcome include brain stem lesions, diffuse axonal injury, and global hypoxia-ischemia. There are some studies on the potential of magnetic resonance spectroscopy (MRS) as a prognostic tool. Biochemical markers in blood and cerebrospinal fluid like neuron-specific enolase (NSE), s100B, and glial fibrillary acidic protein (GFAp) are sensitive to brain damage, but their specificity and predictive power are at present insufficient. To conclude clinical variables and complementary tests, provide a basis for predicting outcome, but no combination of these have been shown to have absolute predictive power.

Conclusions The early stages of unconsciousness and coma can be managed with the application of adequate and rehearsed clinical algorithms as outlined here. The two-track approach (immediate supportive care AND diagnostic forward progress), as well as the multi-professional and team aspects of this work, have herein been emphasized. When severe illness is present which then deteriorates towards permanent brain damage, a more challenging multidimensional clinical situation must be faced. As stated above, even the worst diagnosis of death by neurological criteria in a child need not be a point of total despair to all.

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Acute Pediatric Neurology by Thomas Sejersen, Ching H. Wang


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